Post by Terry S. Singeltary Sr. on Jan 27, 2012 20:41:38 GMT -6
From: Terry S. Singeltary Sr.
Sent: Friday, January 27, 2012 2:06 PM
To: h57@in.gov
Cc: h51@in.gov; h75@in.gov; h53@in.gov; h46@in.gov; h30@in.gov; h54@in.gov; h62@in.gov; h69@in.gov; h64@in.gov; h17@in.gov; h42@in.gov; h56@in.gov
Subject: re-Indiana game farming cervids and CHRONIC WASTING DISEASE THERE FROM
From: Terry S. Singeltary Sr.
Sent: Friday, January 27, 2012 1:56 PM
To: info@indianawildlife.org
Cc: dnrwebmaster@dnr.IN.gov; s1@iga.in.gov; s4@iga.in.gov; h57@in.gov; simpson@indianawildlife.org; twardy@indianawildlife.org; watson@indianawildlife.org
Subject: Re: re-Indiana game farming cervids and CHRONIC WASTING DISEASE THERE FROM House Bill 1265
redo !!!
Subject: re-Indiana game farming cervids and CHRONIC WASTING DISEASE THERE FROM House Bill 1265
Greetings Indiana Government et al ;
re-Indiana game farming cervids and CHRONIC WASTING DISEASE THERE FROM
House Bill 1265, which passed out of the Natural Resources Committee on Tuesday, would make it legal to hunt farm-raised cervidea – deer .....
common sense went out the door long ago $$$
www.in.gov/legislative/bills/2012/IN/IN1265.1.html
be warned, risk your cervid population at your own risks $$$
game farms are nothing more than a petri dish for prion disease and other disease.
DID you see the lates fact that Wisconsin has documented 9 CWD INFECTION GAME FARMS to date, with one of them having the highest CWD infection rate ever, at 80% CWD infection rate.
the highest infection rate to date in the WORLD.
Also, a new study out yesterday on interspecies transmission of CWD and other TSE prion disease, is much easier than first thought.
please see ;
Don’t Forget CWD
Sunday, January 22nd, 2012 | Posted by Dr. Dave Samuel
Nebraska just announced the finding of Chronic Wasting Disease in several counties in the center of the state. This shows the continued spread from western Nebraska. So what you say, “It’s obvious that CWD can’t spread to humans and it sure doesn’t seem to be hurting our deer herds.”
Maybe. Maybe on both counts. The release of information showing the spread of CWD in Nebraska (where I hunt every fall in the eastern part of the state) caused me to check out the website, transmissiblespongiformencephalopathy.blogspot.com/ . That blog lists a lot of other website with tons of information on CWD. CWD is not spread by bacteria but rather by prions that invade and destroy the brain. One very similar disease of humans is Creutzfeldt-Jacob Disease. This blog presents a lot of information on that disease relative to whether humans can get CWD from diseased deer or elk.
Bottom line is that we don’t know a lot about prions. We do know that prions survive in soil for years. We know that deer can be placed in an area where CWD lived and after being removed for over a year, deer placed there again get CWD.
We don’t know how long prions stay viable in the soil. My guess is . . . many, many years. Maybe decades. We know that deer have prions in nervous tissue but also in lymph tissue and even muscles. We know that prions are spread via saliva and urine. (In fact that is why many states are concerned about baiting for deer.) We don’t think prions can be spread via eating deer meat but we aren’t positive. Scary because another prion disease ‘mad cow disease’ can be spread from cows to humans leading to death.
I don’t want to play the ‘what if’ game, but ‘what if’ one hunter got CWD from eating deer meat and that hunter died? Let’s not go there. The ramifications are beyond comprehension.
Another aspect of this problem is found on the above website as well as chronic-wasting-disease.blogspot.com/ . I invite you to go to this website and after you peruse it you will see the relationship of CWD and deer farms. They discuss the Almond deer farm in Wisconsin. A buck shot there in 2002 had CWD and this led to all deer there being killed in 2006. Sixty of the 76 deer killed were CWD positive. In order to keep control of this situation and make sure the fence separating the contaminated farm from wild deer remained intact, the DNR bought the farm in 2011. Remember, this farm probably has CWD prions in the soil, so the potential for spread to wild deer is real. However, 1200 deer outside the farm have been tested and no CWD was found. The DNR wants to make sure the fences stay intact, so they bought it and will do research on CWD there. Seems like a good idea.
A total decontamination of the farm was conducted, but there is no way to know whether prions are still in the soil. They probably are. All timber along the fence is being cut to protect the fence. A second fence 12 feet from the first is being constructed. Apparently the Wisconsin DNR is very concerned about the spread of CWD.
If baiting for deer has been shown to exacerbate the spread of CWD then why do we find baiting being legalized in more and more states? The reason is simple. Hunters want baiting and they apply political pressure to keep baiting if they have it or to get baiting if they do not have it. The DNR in my home state of West Virginia would love to stop baiting for deer to prevent, or slow the spread of CWD from the eastern panhandle of the state, but the hunters won’t let that happen. A political hot potato.
Again, check out the above two web sites then tell me that CWD is not a potential problem for hunters and deer. This ain’t your average disease. So far, humans eating venison are OK and so far deer herds are OK too. But packing large numbers of deer in small areas is an experiment and only time will tell if ‘it’s not nice to fool with Mother Nature.’
bowhunting.net/2012/01/dont-forget-cwd/
CHRONIC WASTING DISEASE CWD WISCONSIN Almond Deer (Buckhorn Flats) Farm Update DECEMBER 2011
Form 1100-001
(R 2/11)
NATURAL RESOURCES BOARD AGENDA ITEM
SUBJECT: Inf01mation Item: Almond Deer Fatm Update
FOR: DECEIVIBER 2011 BOARD MEETING
SNIP...
These laboratory results show that 60 of the 76 animals tested positive for chronic wasting disease. The 76 deer constituted the breeding herd on Hall’s farm. He also operated a hunting preserve on the property until 2005. Four deer, two does and two fawns, the only deer remaining in the former preserve, were killed and tested as well. CWD was not detected in those animals. The total number of deer to test positive from this farm from the initial discovery to final depopulation is 82. The CWD infection rate was nearly 80%, the highest ever in a North American captive herd.
SNIP...
Despite the five year premise plan and site decontamination, The WI DNR has concerns over the bioavailability of infectious prions at this site to wild white-tail deer should these fences be removed. Current research indicates that prions can persist in soil for a minimum of 3 years. However, Georgsson et al. (2006) concluded that prions that produced scrapie disease in sheep remained bioavailable and infectious for at least 16 years in natural Icelandic environments, most likely in contaminated soil. Additionally, the authors reported that from 1978-2004, scrapie recurred on 33 sheep farms, of which 9 recurrences occurred 14-21 years after initial culling and subsequent restocking efforts; these findings further emphasize the effect of environmental contamination on sustaining TSE infectivity and that long-term persistence of prions in soils may be substantially greater than previously thought. Evidence of environmental transmission also was documented in a Colorado research facility where mule deer became infected with CWD in two of three paddocks where infected deer carcasses had decomposed on site 1.8 years earlier, and in one of three paddocks where infected deer had last resided 2.2 years earlier (Miller et al. 2004).
SNIP...
dnr.wi.gov/org/nrboard/2011/december/12-11-2b2.pdf
FULL TEXT AND MORE HERE ;
chronic-wasting-disease.blogspot.com/2011/12/chronic-wasting-disease-cwd-wisconsin.html
Monday, January 16, 2012
9 GAME FARMS IN WISCONSIN TEST POSITIVE FOR CWD
chronic-wasting-disease.blogspot.com/2012/01/9-game-farms-in-wisconsin-test-positive.html
*** Spraker suggested an interesting explanation for the occurrence of CWD. The deer pens at the Foot Hills Campus were built some 30-40 years ago by a Dr. Bob Davis. At or abut that time, allegedly, some scrapie work was conducted at this site. When deer were introduced to the pens they occupied ground that had previously been occupied by sheep.
(PLEASE NOTE SOME OF THESE OLD UK GOVERNMENT FILE URLS ARE SLOW TO OPEN, AND SOMETIMES YOU MAY HAVE TO CLICK ON MULTIPLE TIMES, PLEASE BE PATIENT, ANY PROBLEMS PLEASE WRITE ME PRIVATELY, AND I WILL TRY AND FIX OR SEND YOU OLD PDF FILE...TSS)
collections.europarchive.org/tna/20080102193705/http://www.bseinquiry.gov.uk/files/mb/m11b/tab01.pdf
CWD TSE PRION UPDATE JANUARY 26, 2012
Tuesday, January 24, 2012
CWD found in two free-ranging deer from Macon County Missouri
chronic-wasting-disease.blogspot.com/2012/01/cwd-found-in-two-free-ranging-deer-from.html
Wednesday, January 04, 2012
CWD NEBRASKA NGPC 26 DEER CARCASSES TESTED POSITIVE BUFFALO, CUSTER AND HOLT COUNTIES DURING NOVEMBER HUNT
chronic-wasting-disease.blogspot.com/2012/01/cwd-nebraska-ngpc-26-deer-carcasses.html
Saturday, December 31, 2011
Depopulation Plan Being Developed for Captive Deer Facility in Macon County after second CWD positive confirmation
chronic-wasting-disease.blogspot.com/2011/12/depopulation-plan-being-developed-for.html
Wednesday, December 21, 2011
CWD UTAH San Juan deer hunting unit
chronic-wasting-disease.blogspot.com/2011/12/cwd-utah-san-juan-deer-hunting-unit.html
Thursday, December 29, 2011
Aerosols An underestimated vehicle for transmission of prion diseases?
PRION www.landesbioscience.com please see more on Aerosols and TSE prion disease here ;. ...
transmissiblespongiformencephalopathy.blogspot.com/2011/12/aerosols-underestimated-vehicle-for.html
Sunday, January 22, 2012
Chronic Wasting Disease CWD cervids interspecies transmission
chronic-wasting-disease.blogspot.com/2012/01/chronic-wasting-disease-cwd-cervids.html
Friday, February 25, 2011
Soil clay content underlies prion infection odds
chronic-wasting-disease.blogspot.com/2011/02/soil-clay-content-underlies-prion.html
Wednesday, October 14, 2009
Detection of protease-resistant cervid prion protein in water from a CWD-endemic area
chronic-wasting-disease.blogspot.com/2009/10/detection-of-protease-resistant-cervid.html
CWD TSE prion disease survives ashing to 600 degrees celsius, that’s around 1112 degrees farenheit. you cannot cook the CWD TSE prion disease out of meat. you can take the ash and mix it with saline and inject that ash into a mouse, and the mouse will go down with TSE. Prion Infected Meat-and-Bone Meal Is Still Infectious after Biodiesel Production as well. the TSE prion agent also survives Simulated Wastewater Treatment Processes. IN fact, you should also know that the CWD TSE Prion agent will survive in the environment for years, if not decades. you can bury it and it will not go away. CWD TSE agent is capable of infected your water table i.e. Detection of protease-resistant cervid prion protein in water from a CWD-endemic area. it’s not your ordinary pathogen you can just cook it out and be done with. that’s what’s so worrisome about Iatrogenic mode of transmission, a simple autoclave will not kill this TSE prion agent. atypical strains of TSE prion disease are mounting, and they have been documented to be more virulent, with atypical L-BSE type mad cow having a 50% shorter incubation period than the typical c-BSE. sporadic CJD is NOT, I repeat, sporadic CJD is not a single strain of human TSE, but multiple strains, with atypical strains there from mounting. our hospitals are awash with prion TSE agent. you can take that to the bank. ...
New studies on the heat resistance of hamster-adapted scrapie agent: Threshold survival after ashing at 600°C suggests an inorganic template of replication
Paul Brown*,dagger , Edward H. RauDagger , Bruce K. Johnson*, Alfred E. Bacote*, Clarence J. Gibbs Jr.*, and D. Carleton Gajdusek§ * Laboratory of Central Nervous System Studies, National Institute of Neurological Disorders and Stroke, and Dagger Environmental Protection Branch, Division of Safety, Office of Research Services, National Institutes of Health, Bethesda, MD 20892; and § Institut Alfred Fessard, Centre National de la Recherche Scientifique, 91198 Gif sur Yvette, France Contributed by D. Carleton Gajdusek, December 22, 1999
see full text:
www.pnas.org/cgi/content/full/97/7/3418
Prion Infected Meat-and-Bone Meal Is Still Infectious after Biodiesel Production
Cathrin E. Bruederle,1* Robert M. Hnasko,1 Thomas Kraemer,2 Rafael A. Garcia,3 Michael J. Haas,3 William N. Marmer,3 and John Mark Carter1 1USDA-ARS WRRC, Foodborne Contaminants Research Unit, Albany, California, United States of America 2Forensic Toxicology, Institute of Legal Medicine, Saarland University, Homburg/Saar, Germany 3USDA-ARS ERRC, Fats, Oils and Animal Coproducts Research Unit, Wyndmoor, Pennsylvania, United States of America Neil Mabbott, EditorUniversity of Edinburgh, United Kingdom *
www.ncbi.nlm.nih.gov/pmc/articles/PMC2493038/
Wednesday, October 14, 2009
Detection of protease-resistant cervid prion protein in water from a CWD-endemic area
T.A. Nichols,1,2 Bruce Pulford,1 A. Christy Wyckoff,1,2 Crystal Meyerett,1 Brady Michel,1 Kevin Gertig,3 Edward A. Hoover,1 Jean E. Jewell,4 Glenn C. Telling5 and Mark D. Zabel1,*
1Department of Microbiology, Immunology and Pathology; College of Veterinary Medicine and Biomedical Sciences; Colorado State University; Fort Collins, CO USA; 2National Wildlife Research Center; Wildlife Services; United States Department of Agriculture; Fort Collins, CO USA; 3Fort Collins Utilities; Fort Collins; CO USA; 4Department of Veterinary Sciences; Wyoming State Veterinary Laboratory; University of Wyoming; Laramie, WY USA; 5Department of Microbiology, Immunology, Molecular Genetics and Neurology; Sanders Brown Center on Aging; University of Kentucky; Lexington, KY USA
snip...
The data presented here demonstrate that sPMCA can detect low levels of PrPCWD in the environment, corroborate previous biological and experimental data suggesting long term persistence of prions in the environment2,3 and imply that PrPCWD accumulation over time may contribute to transmission of CWD in areas where it has been endemic for decades. This work demonstrates the utility of sPMCA to evaluate other environmental water sources for PrPCWD, including smaller bodies of water such as vernal pools and wallows, where large numbers of cervids congregate and into which prions from infected animals may be shed and concentrated to infectious levels.
www.landesbioscience.com/journals/prion/NicholsPRION3-3.pdf
OLD BSE TSE PRION HISTORY
The BSE Inquiry / Statement No 19B (supplementary) Dr Alan Colchester Issued 06/08/1999 (not scheduled to give oral evidence) SECOND STATEMENT TO THE BSE INQUIRY Dr A Colchester BA BM BCh PhD FRCP Reader in Neurosciences & Computing, University of Kent at Canterbury; Consultant Neurologist, Guy’s Hospital London and William Harvey Hospital Ashford April 1999
snip...
88. Natural decay: Infectivity persists for a long time in the environment. A study by Palsson in 1979 showed how scrapie was contracted by healthy sheep, after they had grazed on land which had previously been grazed by scrapie-infected sheep, even though the land had lain fallow for three years before the healthy sheep were introduced. Brown also quoted an early experiment of his own (1991), where he had buried scrapie-infected hamster brain and found that he could still detect substantial infectivity three years later near where the material had been placed.
89. Potential environmental routes of infection: Brown discusses the various possible scenarios, including surface or subsurface deposits of TSE-contaminated material, which would lead to a build-up of long-lasting infectivity. Birds feeding on animal remains (such as gulls visiting landfill sites) could disperse infectivity. Other animals could become vectors if they later grazed on contaminated land. "A further question concerns the risk of contamination of the surrounding water table or even surface water channels, by effluents and discarded solid wastes from treatment plants. A reasonable conclusion is that there is a potential for human infection to result from environmental contamination by BSE-infected tissue residues. The potential cannot be quantified because of the huge numbers of uncertainties and assumptions that attend each stage of the disposal process". These comments, from a long established authority on TSEs, closely echo my own statements which were based on a recent examination of all the evidence.
90. Susceptibility: It is likely that transmissibility of the disease to humans in vivo is probably low, because sheep that die from scrapie and cattle that die from BSE are probably a small fraction of the exposed population. However, no definitive data are available.
91. Recommendations for disposal procedures: Brown recommends that material which is actually or potentially contaminated by BSE should be: 1) exposed to caustic soda; 2) thoroughly incinerated under carefully inspected conditions; and 3) that any residue should be buried in landfill, to a depth which would minimise any subsequent animal or human exposure, in areas that would not intersect with any potable water-table source.
snip...
(PLEASE NOTE SOME OF THESE OLD UK GOVERNMENT FILE URLS ARE SLOW TO OPEN, AND SOMETIMES YOU MAY HAVE TO CLICK ON MULTIPLE TIMES, PLEASE BE PATIENT, ANY PROBLEMS PLEASE WRITE ME PRIVATELY, AND I WILL TRY AND FIX OR SEND YOU OLD PDF FILE...TSS)
web.archive.org/web/20030326042814/http://www.bseinquiry.gov.uk/files/ws/s019b.pdf
PAUL BROWN SCRAPIE SOIL TEST
(PLEASE NOTE SOME OF THESE OLD UK GOVERNMENT FILE URLS ARE SLOW TO OPEN, AND SOMETIMES YOU MAY HAVE TO CLICK ON MULTIPLE TIMES, PLEASE BE PATIENT, ANY PROBLEMS PLEASE WRITE ME PRIVATELY, AND I WILL TRY AND FIX OR SEND YOU OLD PDF FILE...TSS)
collections.europarchive.org/tna/20080102120203/http://www.bseinquiry.gov.uk/files/sc/seac07/tab03.pdf
INCINERATION TEMPS
Requirements include:
a. after burning to the range of 800 to 1000*C to eliminate smell; well heck, this is just typical public relations fear factor control. do you actually think they would spend the extra costs for fuel, for such extreme heat, just to eliminate smell, when they spread manure all over your veg's. i think not. what they really meant were any _TSE agents_.
b. Gas scrubbing to eliminate smoke -- though steam may be omitted; c. Stacks to be fitted with grit arreaters;
snip...
1.2 Visual Imact
It is considered that the requirement for any carcase incinerator disign would be to ensure that the operations relating to the reception, storage and decepitation of diseased carcasses must not be publicly visible and that any part of a carcase could not be removed or interfered with by animals or birds.
full text;
web.archive.org/web/20040625024009/http://www.bseinquiry.gov.uk/files/yb/1989/04/03006001.pdf
18. The EA’s assertion at the Thruxted planning inquiry that the precautionary principle does not apply in the case of Thruxted Mill in view of the low risk entailed by its effluent discharge is entirely unfounded. The source data presented by the EA at the Thruxted Inquiry derive in part from its assumptions concerning the segregation of infectivity to the various products of rendering. The EA also stipulates a minimum particle size of 1013 molecules for human infection and assumes there is a 2500-fold reduction of infectivity by rendering, filtration and biological treatment prior to discharge. In fact, the minimum particle size may be at least 1012 times lower. The reduction in the input levels of BSE infectivity prior to discharge will also be very substantially less than implied in the EA source data, and may indeed be minimal. The EA assumes that biological treatment of the rendering effluent will reduce or eliminate BSE infectivity. This is probably the exact opposite of what is actually likely to happen.
19. In his proof of evidence at the Thruxted Inquiry, Mr Young asserted that "effective filtering of clumps of material is likely". As already mentioned, infectious prions are known to pass
web.archive.org/web/20040625025024/http://www.bseinquiry.gov.uk/files/ws/s018.pdf
[PDF]BSE INQUIRY Statement of behalf of the Environment Agency ...
... his Statement of March 1998 to the BSE Inquiry ... systems subject to regular or intermittent contamination by rapid movement of recharge water ...
BSE INQUIRY
Statement of behalf of the Environment Agency
Concerning Thruxted Mill
By
Mr C. P. Young
Principal Hydrogeologist, Soil Waste and Groundwater Group
WRc plc; Medmenham, Bucks
web.archive.org/web/20040625023104/http://www.bseinquiry.gov.uk/files/ws/s490.pdf
Friday, February 25, 2011
Soil clay content underlies prion infection odds
www.nature.com/ncomms/journal/v2/n2/full/ncomms1203.html
chronic-wasting-disease.blogspot.com/2011/02/soil-clay-content-underlies-prion.html
UPDATED DATA ON 2ND CWD STRAIN
Wednesday, September 08, 2010
CWD PRION CONGRESS SEPTEMBER 8-11 2010
chronic-wasting-disease.blogspot.com/2010/09/cwd-prion-2010.html
Monday, August 8, 2011
Susceptibility of Domestic Cats to CWD Infection
Oral.29: Susceptibility of Domestic Cats to CWD Infection
felinespongiformencephalopathyfse.blogspot.com/
Sunday, January 22, 2012
Chronic Wasting Disease CWD cervids interspecies transmission
chronic-wasting-disease.blogspot.com/2012/01/chronic-wasting-disease-cwd-cervids.html
Thursday, February 17, 2011
Environmental Sources of Scrapie Prions
scrapie-usa.blogspot.com/2011/02/environmental-sources-of-scrapie-prions.html
Wednesday, January 18, 2012
BSE IN GOATS CAN BE MISTAKEN FOR SCRAPIE
February 1, 2012
transmissiblespongiformencephalopathy.blogspot.com/2012/01/bse-in-goats-can-be-mistaken-for.html
chronic-wasting-disease.blogspot.com/
transmissiblespongiformencephalopathy.blogspot.com/
these blogs are for educational use about PrPSc Prion TSE disease, so that folks will become more aware, enhance debate, and awareness. ...
kind regards,
terry
Sent: Friday, January 27, 2012 2:06 PM
To: h57@in.gov
Cc: h51@in.gov; h75@in.gov; h53@in.gov; h46@in.gov; h30@in.gov; h54@in.gov; h62@in.gov; h69@in.gov; h64@in.gov; h17@in.gov; h42@in.gov; h56@in.gov
Subject: re-Indiana game farming cervids and CHRONIC WASTING DISEASE THERE FROM
From: Terry S. Singeltary Sr.
Sent: Friday, January 27, 2012 1:56 PM
To: info@indianawildlife.org
Cc: dnrwebmaster@dnr.IN.gov; s1@iga.in.gov; s4@iga.in.gov; h57@in.gov; simpson@indianawildlife.org; twardy@indianawildlife.org; watson@indianawildlife.org
Subject: Re: re-Indiana game farming cervids and CHRONIC WASTING DISEASE THERE FROM House Bill 1265
redo !!!
Subject: re-Indiana game farming cervids and CHRONIC WASTING DISEASE THERE FROM House Bill 1265
Greetings Indiana Government et al ;
re-Indiana game farming cervids and CHRONIC WASTING DISEASE THERE FROM
House Bill 1265, which passed out of the Natural Resources Committee on Tuesday, would make it legal to hunt farm-raised cervidea – deer .....
common sense went out the door long ago $$$
www.in.gov/legislative/bills/2012/IN/IN1265.1.html
be warned, risk your cervid population at your own risks $$$
game farms are nothing more than a petri dish for prion disease and other disease.
DID you see the lates fact that Wisconsin has documented 9 CWD INFECTION GAME FARMS to date, with one of them having the highest CWD infection rate ever, at 80% CWD infection rate.
the highest infection rate to date in the WORLD.
Also, a new study out yesterday on interspecies transmission of CWD and other TSE prion disease, is much easier than first thought.
please see ;
Don’t Forget CWD
Sunday, January 22nd, 2012 | Posted by Dr. Dave Samuel
Nebraska just announced the finding of Chronic Wasting Disease in several counties in the center of the state. This shows the continued spread from western Nebraska. So what you say, “It’s obvious that CWD can’t spread to humans and it sure doesn’t seem to be hurting our deer herds.”
Maybe. Maybe on both counts. The release of information showing the spread of CWD in Nebraska (where I hunt every fall in the eastern part of the state) caused me to check out the website, transmissiblespongiformencephalopathy.blogspot.com/ . That blog lists a lot of other website with tons of information on CWD. CWD is not spread by bacteria but rather by prions that invade and destroy the brain. One very similar disease of humans is Creutzfeldt-Jacob Disease. This blog presents a lot of information on that disease relative to whether humans can get CWD from diseased deer or elk.
Bottom line is that we don’t know a lot about prions. We do know that prions survive in soil for years. We know that deer can be placed in an area where CWD lived and after being removed for over a year, deer placed there again get CWD.
We don’t know how long prions stay viable in the soil. My guess is . . . many, many years. Maybe decades. We know that deer have prions in nervous tissue but also in lymph tissue and even muscles. We know that prions are spread via saliva and urine. (In fact that is why many states are concerned about baiting for deer.) We don’t think prions can be spread via eating deer meat but we aren’t positive. Scary because another prion disease ‘mad cow disease’ can be spread from cows to humans leading to death.
I don’t want to play the ‘what if’ game, but ‘what if’ one hunter got CWD from eating deer meat and that hunter died? Let’s not go there. The ramifications are beyond comprehension.
Another aspect of this problem is found on the above website as well as chronic-wasting-disease.blogspot.com/ . I invite you to go to this website and after you peruse it you will see the relationship of CWD and deer farms. They discuss the Almond deer farm in Wisconsin. A buck shot there in 2002 had CWD and this led to all deer there being killed in 2006. Sixty of the 76 deer killed were CWD positive. In order to keep control of this situation and make sure the fence separating the contaminated farm from wild deer remained intact, the DNR bought the farm in 2011. Remember, this farm probably has CWD prions in the soil, so the potential for spread to wild deer is real. However, 1200 deer outside the farm have been tested and no CWD was found. The DNR wants to make sure the fences stay intact, so they bought it and will do research on CWD there. Seems like a good idea.
A total decontamination of the farm was conducted, but there is no way to know whether prions are still in the soil. They probably are. All timber along the fence is being cut to protect the fence. A second fence 12 feet from the first is being constructed. Apparently the Wisconsin DNR is very concerned about the spread of CWD.
If baiting for deer has been shown to exacerbate the spread of CWD then why do we find baiting being legalized in more and more states? The reason is simple. Hunters want baiting and they apply political pressure to keep baiting if they have it or to get baiting if they do not have it. The DNR in my home state of West Virginia would love to stop baiting for deer to prevent, or slow the spread of CWD from the eastern panhandle of the state, but the hunters won’t let that happen. A political hot potato.
Again, check out the above two web sites then tell me that CWD is not a potential problem for hunters and deer. This ain’t your average disease. So far, humans eating venison are OK and so far deer herds are OK too. But packing large numbers of deer in small areas is an experiment and only time will tell if ‘it’s not nice to fool with Mother Nature.’
bowhunting.net/2012/01/dont-forget-cwd/
CHRONIC WASTING DISEASE CWD WISCONSIN Almond Deer (Buckhorn Flats) Farm Update DECEMBER 2011
Form 1100-001
(R 2/11)
NATURAL RESOURCES BOARD AGENDA ITEM
SUBJECT: Inf01mation Item: Almond Deer Fatm Update
FOR: DECEIVIBER 2011 BOARD MEETING
SNIP...
These laboratory results show that 60 of the 76 animals tested positive for chronic wasting disease. The 76 deer constituted the breeding herd on Hall’s farm. He also operated a hunting preserve on the property until 2005. Four deer, two does and two fawns, the only deer remaining in the former preserve, were killed and tested as well. CWD was not detected in those animals. The total number of deer to test positive from this farm from the initial discovery to final depopulation is 82. The CWD infection rate was nearly 80%, the highest ever in a North American captive herd.
SNIP...
Despite the five year premise plan and site decontamination, The WI DNR has concerns over the bioavailability of infectious prions at this site to wild white-tail deer should these fences be removed. Current research indicates that prions can persist in soil for a minimum of 3 years. However, Georgsson et al. (2006) concluded that prions that produced scrapie disease in sheep remained bioavailable and infectious for at least 16 years in natural Icelandic environments, most likely in contaminated soil. Additionally, the authors reported that from 1978-2004, scrapie recurred on 33 sheep farms, of which 9 recurrences occurred 14-21 years after initial culling and subsequent restocking efforts; these findings further emphasize the effect of environmental contamination on sustaining TSE infectivity and that long-term persistence of prions in soils may be substantially greater than previously thought. Evidence of environmental transmission also was documented in a Colorado research facility where mule deer became infected with CWD in two of three paddocks where infected deer carcasses had decomposed on site 1.8 years earlier, and in one of three paddocks where infected deer had last resided 2.2 years earlier (Miller et al. 2004).
SNIP...
dnr.wi.gov/org/nrboard/2011/december/12-11-2b2.pdf
FULL TEXT AND MORE HERE ;
chronic-wasting-disease.blogspot.com/2011/12/chronic-wasting-disease-cwd-wisconsin.html
Monday, January 16, 2012
9 GAME FARMS IN WISCONSIN TEST POSITIVE FOR CWD
chronic-wasting-disease.blogspot.com/2012/01/9-game-farms-in-wisconsin-test-positive.html
*** Spraker suggested an interesting explanation for the occurrence of CWD. The deer pens at the Foot Hills Campus were built some 30-40 years ago by a Dr. Bob Davis. At or abut that time, allegedly, some scrapie work was conducted at this site. When deer were introduced to the pens they occupied ground that had previously been occupied by sheep.
(PLEASE NOTE SOME OF THESE OLD UK GOVERNMENT FILE URLS ARE SLOW TO OPEN, AND SOMETIMES YOU MAY HAVE TO CLICK ON MULTIPLE TIMES, PLEASE BE PATIENT, ANY PROBLEMS PLEASE WRITE ME PRIVATELY, AND I WILL TRY AND FIX OR SEND YOU OLD PDF FILE...TSS)
collections.europarchive.org/tna/20080102193705/http://www.bseinquiry.gov.uk/files/mb/m11b/tab01.pdf
CWD TSE PRION UPDATE JANUARY 26, 2012
Tuesday, January 24, 2012
CWD found in two free-ranging deer from Macon County Missouri
chronic-wasting-disease.blogspot.com/2012/01/cwd-found-in-two-free-ranging-deer-from.html
Wednesday, January 04, 2012
CWD NEBRASKA NGPC 26 DEER CARCASSES TESTED POSITIVE BUFFALO, CUSTER AND HOLT COUNTIES DURING NOVEMBER HUNT
chronic-wasting-disease.blogspot.com/2012/01/cwd-nebraska-ngpc-26-deer-carcasses.html
Saturday, December 31, 2011
Depopulation Plan Being Developed for Captive Deer Facility in Macon County after second CWD positive confirmation
chronic-wasting-disease.blogspot.com/2011/12/depopulation-plan-being-developed-for.html
Wednesday, December 21, 2011
CWD UTAH San Juan deer hunting unit
chronic-wasting-disease.blogspot.com/2011/12/cwd-utah-san-juan-deer-hunting-unit.html
Thursday, December 29, 2011
Aerosols An underestimated vehicle for transmission of prion diseases?
PRION www.landesbioscience.com please see more on Aerosols and TSE prion disease here ;. ...
transmissiblespongiformencephalopathy.blogspot.com/2011/12/aerosols-underestimated-vehicle-for.html
Sunday, January 22, 2012
Chronic Wasting Disease CWD cervids interspecies transmission
chronic-wasting-disease.blogspot.com/2012/01/chronic-wasting-disease-cwd-cervids.html
Friday, February 25, 2011
Soil clay content underlies prion infection odds
chronic-wasting-disease.blogspot.com/2011/02/soil-clay-content-underlies-prion.html
Wednesday, October 14, 2009
Detection of protease-resistant cervid prion protein in water from a CWD-endemic area
chronic-wasting-disease.blogspot.com/2009/10/detection-of-protease-resistant-cervid.html
CWD TSE prion disease survives ashing to 600 degrees celsius, that’s around 1112 degrees farenheit. you cannot cook the CWD TSE prion disease out of meat. you can take the ash and mix it with saline and inject that ash into a mouse, and the mouse will go down with TSE. Prion Infected Meat-and-Bone Meal Is Still Infectious after Biodiesel Production as well. the TSE prion agent also survives Simulated Wastewater Treatment Processes. IN fact, you should also know that the CWD TSE Prion agent will survive in the environment for years, if not decades. you can bury it and it will not go away. CWD TSE agent is capable of infected your water table i.e. Detection of protease-resistant cervid prion protein in water from a CWD-endemic area. it’s not your ordinary pathogen you can just cook it out and be done with. that’s what’s so worrisome about Iatrogenic mode of transmission, a simple autoclave will not kill this TSE prion agent. atypical strains of TSE prion disease are mounting, and they have been documented to be more virulent, with atypical L-BSE type mad cow having a 50% shorter incubation period than the typical c-BSE. sporadic CJD is NOT, I repeat, sporadic CJD is not a single strain of human TSE, but multiple strains, with atypical strains there from mounting. our hospitals are awash with prion TSE agent. you can take that to the bank. ...
New studies on the heat resistance of hamster-adapted scrapie agent: Threshold survival after ashing at 600°C suggests an inorganic template of replication
Paul Brown*,dagger , Edward H. RauDagger , Bruce K. Johnson*, Alfred E. Bacote*, Clarence J. Gibbs Jr.*, and D. Carleton Gajdusek§ * Laboratory of Central Nervous System Studies, National Institute of Neurological Disorders and Stroke, and Dagger Environmental Protection Branch, Division of Safety, Office of Research Services, National Institutes of Health, Bethesda, MD 20892; and § Institut Alfred Fessard, Centre National de la Recherche Scientifique, 91198 Gif sur Yvette, France Contributed by D. Carleton Gajdusek, December 22, 1999
see full text:
www.pnas.org/cgi/content/full/97/7/3418
Prion Infected Meat-and-Bone Meal Is Still Infectious after Biodiesel Production
Cathrin E. Bruederle,1* Robert M. Hnasko,1 Thomas Kraemer,2 Rafael A. Garcia,3 Michael J. Haas,3 William N. Marmer,3 and John Mark Carter1 1USDA-ARS WRRC, Foodborne Contaminants Research Unit, Albany, California, United States of America 2Forensic Toxicology, Institute of Legal Medicine, Saarland University, Homburg/Saar, Germany 3USDA-ARS ERRC, Fats, Oils and Animal Coproducts Research Unit, Wyndmoor, Pennsylvania, United States of America Neil Mabbott, EditorUniversity of Edinburgh, United Kingdom *
www.ncbi.nlm.nih.gov/pmc/articles/PMC2493038/
Wednesday, October 14, 2009
Detection of protease-resistant cervid prion protein in water from a CWD-endemic area
T.A. Nichols,1,2 Bruce Pulford,1 A. Christy Wyckoff,1,2 Crystal Meyerett,1 Brady Michel,1 Kevin Gertig,3 Edward A. Hoover,1 Jean E. Jewell,4 Glenn C. Telling5 and Mark D. Zabel1,*
1Department of Microbiology, Immunology and Pathology; College of Veterinary Medicine and Biomedical Sciences; Colorado State University; Fort Collins, CO USA; 2National Wildlife Research Center; Wildlife Services; United States Department of Agriculture; Fort Collins, CO USA; 3Fort Collins Utilities; Fort Collins; CO USA; 4Department of Veterinary Sciences; Wyoming State Veterinary Laboratory; University of Wyoming; Laramie, WY USA; 5Department of Microbiology, Immunology, Molecular Genetics and Neurology; Sanders Brown Center on Aging; University of Kentucky; Lexington, KY USA
snip...
The data presented here demonstrate that sPMCA can detect low levels of PrPCWD in the environment, corroborate previous biological and experimental data suggesting long term persistence of prions in the environment2,3 and imply that PrPCWD accumulation over time may contribute to transmission of CWD in areas where it has been endemic for decades. This work demonstrates the utility of sPMCA to evaluate other environmental water sources for PrPCWD, including smaller bodies of water such as vernal pools and wallows, where large numbers of cervids congregate and into which prions from infected animals may be shed and concentrated to infectious levels.
www.landesbioscience.com/journals/prion/NicholsPRION3-3.pdf
OLD BSE TSE PRION HISTORY
The BSE Inquiry / Statement No 19B (supplementary) Dr Alan Colchester Issued 06/08/1999 (not scheduled to give oral evidence) SECOND STATEMENT TO THE BSE INQUIRY Dr A Colchester BA BM BCh PhD FRCP Reader in Neurosciences & Computing, University of Kent at Canterbury; Consultant Neurologist, Guy’s Hospital London and William Harvey Hospital Ashford April 1999
snip...
88. Natural decay: Infectivity persists for a long time in the environment. A study by Palsson in 1979 showed how scrapie was contracted by healthy sheep, after they had grazed on land which had previously been grazed by scrapie-infected sheep, even though the land had lain fallow for three years before the healthy sheep were introduced. Brown also quoted an early experiment of his own (1991), where he had buried scrapie-infected hamster brain and found that he could still detect substantial infectivity three years later near where the material had been placed.
89. Potential environmental routes of infection: Brown discusses the various possible scenarios, including surface or subsurface deposits of TSE-contaminated material, which would lead to a build-up of long-lasting infectivity. Birds feeding on animal remains (such as gulls visiting landfill sites) could disperse infectivity. Other animals could become vectors if they later grazed on contaminated land. "A further question concerns the risk of contamination of the surrounding water table or even surface water channels, by effluents and discarded solid wastes from treatment plants. A reasonable conclusion is that there is a potential for human infection to result from environmental contamination by BSE-infected tissue residues. The potential cannot be quantified because of the huge numbers of uncertainties and assumptions that attend each stage of the disposal process". These comments, from a long established authority on TSEs, closely echo my own statements which were based on a recent examination of all the evidence.
90. Susceptibility: It is likely that transmissibility of the disease to humans in vivo is probably low, because sheep that die from scrapie and cattle that die from BSE are probably a small fraction of the exposed population. However, no definitive data are available.
91. Recommendations for disposal procedures: Brown recommends that material which is actually or potentially contaminated by BSE should be: 1) exposed to caustic soda; 2) thoroughly incinerated under carefully inspected conditions; and 3) that any residue should be buried in landfill, to a depth which would minimise any subsequent animal or human exposure, in areas that would not intersect with any potable water-table source.
snip...
(PLEASE NOTE SOME OF THESE OLD UK GOVERNMENT FILE URLS ARE SLOW TO OPEN, AND SOMETIMES YOU MAY HAVE TO CLICK ON MULTIPLE TIMES, PLEASE BE PATIENT, ANY PROBLEMS PLEASE WRITE ME PRIVATELY, AND I WILL TRY AND FIX OR SEND YOU OLD PDF FILE...TSS)
web.archive.org/web/20030326042814/http://www.bseinquiry.gov.uk/files/ws/s019b.pdf
PAUL BROWN SCRAPIE SOIL TEST
(PLEASE NOTE SOME OF THESE OLD UK GOVERNMENT FILE URLS ARE SLOW TO OPEN, AND SOMETIMES YOU MAY HAVE TO CLICK ON MULTIPLE TIMES, PLEASE BE PATIENT, ANY PROBLEMS PLEASE WRITE ME PRIVATELY, AND I WILL TRY AND FIX OR SEND YOU OLD PDF FILE...TSS)
collections.europarchive.org/tna/20080102120203/http://www.bseinquiry.gov.uk/files/sc/seac07/tab03.pdf
INCINERATION TEMPS
Requirements include:
a. after burning to the range of 800 to 1000*C to eliminate smell; well heck, this is just typical public relations fear factor control. do you actually think they would spend the extra costs for fuel, for such extreme heat, just to eliminate smell, when they spread manure all over your veg's. i think not. what they really meant were any _TSE agents_.
b. Gas scrubbing to eliminate smoke -- though steam may be omitted; c. Stacks to be fitted with grit arreaters;
snip...
1.2 Visual Imact
It is considered that the requirement for any carcase incinerator disign would be to ensure that the operations relating to the reception, storage and decepitation of diseased carcasses must not be publicly visible and that any part of a carcase could not be removed or interfered with by animals or birds.
full text;
web.archive.org/web/20040625024009/http://www.bseinquiry.gov.uk/files/yb/1989/04/03006001.pdf
18. The EA’s assertion at the Thruxted planning inquiry that the precautionary principle does not apply in the case of Thruxted Mill in view of the low risk entailed by its effluent discharge is entirely unfounded. The source data presented by the EA at the Thruxted Inquiry derive in part from its assumptions concerning the segregation of infectivity to the various products of rendering. The EA also stipulates a minimum particle size of 1013 molecules for human infection and assumes there is a 2500-fold reduction of infectivity by rendering, filtration and biological treatment prior to discharge. In fact, the minimum particle size may be at least 1012 times lower. The reduction in the input levels of BSE infectivity prior to discharge will also be very substantially less than implied in the EA source data, and may indeed be minimal. The EA assumes that biological treatment of the rendering effluent will reduce or eliminate BSE infectivity. This is probably the exact opposite of what is actually likely to happen.
19. In his proof of evidence at the Thruxted Inquiry, Mr Young asserted that "effective filtering of clumps of material is likely". As already mentioned, infectious prions are known to pass
web.archive.org/web/20040625025024/http://www.bseinquiry.gov.uk/files/ws/s018.pdf
[PDF]BSE INQUIRY Statement of behalf of the Environment Agency ...
... his Statement of March 1998 to the BSE Inquiry ... systems subject to regular or intermittent contamination by rapid movement of recharge water ...
BSE INQUIRY
Statement of behalf of the Environment Agency
Concerning Thruxted Mill
By
Mr C. P. Young
Principal Hydrogeologist, Soil Waste and Groundwater Group
WRc plc; Medmenham, Bucks
web.archive.org/web/20040625023104/http://www.bseinquiry.gov.uk/files/ws/s490.pdf
Friday, February 25, 2011
Soil clay content underlies prion infection odds
www.nature.com/ncomms/journal/v2/n2/full/ncomms1203.html
chronic-wasting-disease.blogspot.com/2011/02/soil-clay-content-underlies-prion.html
UPDATED DATA ON 2ND CWD STRAIN
Wednesday, September 08, 2010
CWD PRION CONGRESS SEPTEMBER 8-11 2010
chronic-wasting-disease.blogspot.com/2010/09/cwd-prion-2010.html
Monday, August 8, 2011
Susceptibility of Domestic Cats to CWD Infection
Oral.29: Susceptibility of Domestic Cats to CWD Infection
felinespongiformencephalopathyfse.blogspot.com/
Sunday, January 22, 2012
Chronic Wasting Disease CWD cervids interspecies transmission
chronic-wasting-disease.blogspot.com/2012/01/chronic-wasting-disease-cwd-cervids.html
Thursday, February 17, 2011
Environmental Sources of Scrapie Prions
scrapie-usa.blogspot.com/2011/02/environmental-sources-of-scrapie-prions.html
Wednesday, January 18, 2012
BSE IN GOATS CAN BE MISTAKEN FOR SCRAPIE
February 1, 2012
transmissiblespongiformencephalopathy.blogspot.com/2012/01/bse-in-goats-can-be-mistaken-for.html
chronic-wasting-disease.blogspot.com/
transmissiblespongiformencephalopathy.blogspot.com/
these blogs are for educational use about PrPSc Prion TSE disease, so that folks will become more aware, enhance debate, and awareness. ...
kind regards,
terry
I could not agree more. ...tss
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